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Several of the neurodegenerative and neuroinflammatory biomarkers were higher in people whose p-tau/Aβ42 ratio indicated they had AD pathology than in those who did not. This was true for NfL, neurogranin, α-synuclein, YKL-40, S100b, and GFAP in people with MCI due to AD, and only true for neurogranin in cognitively normal people. These same ...
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